FEATURES

DESCRIPTION

Also known as SG101_ARATH, SAG101. Acyl hydrolase that triggers the leaf senescence onset. Can use triolein as substrate to produce oleic acids., Involved in the EDS1-dependent intrinsic and indispensable resistance signaling pathway; together with PAD4, required for programmed cell death triggered by RPS4 in response to avirulent pathogens (e.g. P.syringae pv. tomato strain DC3000 and H.parasitica isolates CALA2 and EMWA1) and in restricting the growth of virulent pathogens (e.g. H.parasitica isolates NOCO2 and P.syringae pv. tomato strain DC3000 avrRps4). Contributes in reinforcing the immune response around hypersensitive response foci (PubMed:21434927). Regulates the nuclear localization of EDS1. Essential for the RPP8/HRT-mediated resistance to the turnip crinkle virus (TCV). Involved in the post-invasion resistance to P.pachyrhizi in the mesophyll. Part of a nuclear complex made of EDS1, PAD4 and SAG101, that can be redirected to the cytoplasm in the presence of an extranuclear form of EDS1 (PubMed:22072959). Interacts directly with EDS1.

EXTERNAL LINKS

SAG101

Senescence-associated carboxylesterase 101

SAG101

Molecular Synopsis